Okay, guys, the DR blog has been absolutely flooded with Stained posts as of late, and I think many of us can agree that this is a problem. I put some serious thought into it, and I think I figured out the solution, so I called an old friend. It only took the two low payments of $19.95 and the sacrifice of a virgin, so here here he is.
Scientists Discover That Eyes Are Windows To The Soul
The eye is the window to the universe, and some would say they are also windows to the soul… We have heard this phrase get passed around before: “The eyes are the windows of the soul”. People usually say this when they can see pain, anger, or some other emotion in somebody else’s eyes. But recent research gives a whole new meaning to this phrase. Eyes not only windows to emotions, they are windows to the soul.
TFW people complaining about stained being a gem gene but one of the gem genes is a gradient
Do you know how hard it is to make the gradient for Iri / shim? Not at all if you know what you’re doing. I could whip out a new color every 2-5 minutes if I got in to the groove of it. Most of that time would be making sure the color fits the style sheet.
The funny thing is that iri shim on the basic breeds looks like it had MORE work put in to them and they look UGLY as heck compared to iri/shim on newer breeds. Time put in /=/ how good it looks, sometimes simpler (and thus quicker/less work!) is better.
I don’t like stained and honestly I’m never going to buy it but people complaining about stained but turning around and praising iri / shim like it’s way harder to make then stained are kind are over estimating how hard it is to make a smooth gradient.
TLDR; If time put in = if it should be a gem gene or not then iri / shim should not be a gem gene because it takes 2-3 minutes of more work to make than stained. (honestly wouldn’t complain if it wasn’t)
Our engineering team has been working on new tools to help us generate and implement genes in a much faster and more efficient manner that does not sacrifice the image and color quality we’ve come to value as part of our site’s style. These tools are being developed to make future gene implementation much easier, but also come with the added benefit of allowing us to expand our color wheel without also increasing artist workload exponentially.
We are currently working on converting our existing breed art templates into ones that will be compatible with our new tools. Every time we finish 10, we will be revealing a color. Some will be old, some will be new.
To answer your questions:
We are intending to only expand the color wheel once, as we would like to minimize the disruption to player’s breeding ranges.
New colors will go between existing colors on the wheel so that they are in places that make sense for their range. We will not reshuffle the wheel, and your ranges will remain close to the same, but expanded.
New colors will ONLY be able to be bred, hatched, and scattered for.
Example: If you had a Rose to Magenta range, you’re not suddenly going to have a green in there. It will be more pinks.
To remain fair to all of our players, only the original 67 colors will be available during account registration and new dragon creation.
In April 2015, a paper by Chinese scientists about their attempts to edit the DNA of a human embryo rocked the scientific world and set off a furious debate. Leading scientists warned that altering the human germ line without studying the consequences could have horrific consequences. Geneticists with good intentions could mistakenly engineer changes in DNA that generate dangerous mutations and cause painful deaths. Scientists — and countries — with less noble intentions could again try to build a race of superhumans.
Serotonin is one of the major neurotransmitters (i.e. chemicals) in
the brain. It’s very connected to our emotions and so it’s not a
coincidence that a lot of the drugs that are used to treat depression
and anxiety act on the serotonin system in the brain. This is clearly a
very important chemical for determining the nature of our emotional
The serotonin transporter gene
is involved with the regulation of serotonin in the brain. People are born with variations of
this gene. The long variation (or “allele”) clears serotonin out of the neural synapse
more efficiently. The short variation is less efficient, which lets the
serotonin hang around a little bit longer in the synapse.
The short variation was originally considered a risk gene because it was associated with depression and anxiety — but it’s now being thought of as a sensitivity gene.
There is enough DNA in an average person’s body to stretch from the Sun to Pluto and back, 17 times.
The human genome, the genetic code in each human cell, contains 23 DNA molecules each containing from 500 thousand to 2.5 million nucleotide pairs. DNA molecules of this size are 1.7 to 8.5 cm long when uncoiled, or about 5 cm on average. There are about 37 trillion cells in the human body and if you’d uncoil all of the DNA encased in each cell and put them end to end, then these would sum to a total length of 2×1014 meters or enough for 17 Pluto roundtrips (1.2×1013 meters/Pluto roundtrip).
Elephants have evolved extra copies of a gene that fights tumour cells, according to two independent studies1, 2 — offering an explanation for why the animals so rarely develop cancer.
Why elephants do not get cancer is a famous conundrum that was posed — in a different form — by epidemiologist Richard Peto of the University of Oxford, UK, in the 1970s3.
Peto noted that, in general, there is little relationship between
cancer rates and the body size or age of animals. That is surprising:
the cells of large-bodied or older animals should have divided many more
times than those of smaller or younger ones, so should possess more
random mutations predisposing them to cancer. Peto speculated that there
might be an intrinsic biological mechanism that protects cells from
cancer as they age and expand.
At least one
solution to Peto’s paradox may now have been found, according to a pair
of papers independently published this week. Elephants have 20 copies of
a gene called p53 (or, more properly, TP53), in their
genome, where humans and other mammals have only one. The gene is known
as a tumour suppressor, and it snaps to action when cells suffer DNA
damage, churning out copies of its associated p53 protein and either
repairing the damage or killing off the cell.