As a teacher, if a child hits their head, I automatically have the nurse check the child out. Sometimes with head injuries you can't see something that is wrong right away. The parent is always notified. If the child had a medical condition prior to the injury, we'd probably ask if the parents wanted to take the child to the doctor OR depending on the case we'd call 911. I want to know why Sandra Bland, who stated on tape that she has epilepsy and that her head had been hit, was not given proper medical attention. I read the jailing report. There is no mention of her receiving medical care.
Parents of baby Mary Jane Pierce win temporary injunction to keep her on life-support
A Chilliwack couple fighting to keep their four-month-old baby alive won a temporary injunction Tuesday at B.C. Supreme Court to stop doctors from removing life-support.

A lot of nope on the part of the government here, not least the refusal to take responsibility for what they’re trying to do. But there’s also ableism regarding both the baby (remove life support) and the parents (they have epilepsy and apparently that means the government doesn’t think they’re able to be good parents).

She was born prematurely, and she will have a hard life (and has already had a hard life), but she is currently alive and her parents want her. There shouldn’t be a question about whether she stays on life support or whether her parents keep her.


Hi guys. My name is Lex and I have intractable juvenile myoclonic epilepsy along with a long list of other disorder and diseases. I take around 40-60 pills every day with weekly IV infusions plus IVIG every other month. I go to the emergency room on average twice a week. I have been a critical patient
more than ten time and have survived many visits to the ICU. The doctor’s themselves think it’s a miracle I can talk nonetheless breathing.

I am not asking for money, reblogs, or likes but prayers. The biggest gift you can give me is lighting a candle in for me.

I am not afraid of death, but there are so many more things I plan to do in my life. Right now I just want to live long enough to kiss my boyfriend in the airport. I don’t plan on dying till then but I need your help of prayers and thoughts. To be extra amazing though please go see if you are a candidate to be a donor and donate blood. Donor blood has saved me along with millions of other people. You don’t always have to give money to be a hero.

Thank you,

Epilepsy warning for the movie Tomorrowland!

If you are epileptic, I strongly suggest you do not see the movie Tomorrowland. Dont get me wrong, I just saw the movie and loved it, but i noticed there are quite a few parts with many flashing lights and other things that could potentially harm someone with epilepsy. If someone has better information about this, please add onto this. Please stay safe guys!

Quiet that Ringing in the Brain

A new drug that selectively affects potassium channels in the brain may offer effective treatment for epilepsy and prevent tinnitus, UConn neurophysiologist Anastasios Tzingounis and colleagues report in the June 10 Journal of Neuroscience.

Epilepsy and tinnitus are both caused by overly excitable nerve cells. Healthy nerves have a built-in system that slams on the brakes when they get too excited. But in some people this braking system doesn’t work, and the nerves run amok, signaling so much that the brain gets overloaded and has a seizure (epilepsy) or hears phantom ringing (tinnitus). About 65 million people worldwide are affected by epilepsy. The numbers on tinnitus are not as clear-cut, but the American Tinnitus Association estimates 2 million people have tinnitus so disabling they have trouble functioning in daily life.

The existing drugs to treat epilepsy don’t always work, and can have serious side effects. One of the more effective, called retigabine, helps open KCNQ potassium channels, which are the “brakes” that shut down the signaling of overly excited nerves. Unfortunately, retigabine has significant adverse side effects, including sleepiness, dizziness, problems with urination and hearing, and an unnerving tendency to turn people’s skin and eyes blue. Because of this, it’s usually only given to adults who don’t get relief from other epilepsy drugs.

Tzingounis’s research focuses on KCNQ potassium channels and how they work. He became interested in the topic several years ago, when doctors around the world began reporting infants with severe, brain-damaging seizures. Genetic testing showed that the children with this problem had genetic differences in their KCNQ potassium channels. Most existing anti-seizure drugs don’t work for these children, and few physicians are willing to prescribe retigabine for babies because of its side effects.

A neurobiologist at the University of Pittsburgh, Thanos Tzounopoulos, who specializes in tinnitus and knew about Tzingounis’s work on potassium channels, contacted Tzingounis in 2013 and asked if he’d like to test out a new drug candidate. The drug, SF0034, was chemically identical to retigabine, except that it had an extra fluorine atom. A company called SciFluor had developed SF0034, and wanted to know whether the compound had promise against epilepsy and tinnitus. The two researchers thought the drug had the potential to be much better than retigabine, and began working together to test it.

The most important question to answer was whether SF0034 works on KCNQ potassium channels the same way retigabine does, and if so, was it better or worse than its parent compound?

KCNQ potassium channels are found in the initial segment of axons, long nerve fibers that reach out and almost, but don’t quite, touch other cells. The gap between the axon and the other cell is called a synapse. When the cell wants to signal to the axon, it floods the synapse with sodium ions to create an electrical potential. When that electrical potential goes on too long, or gets out of hand, the KCNQ potassium channel kicks in. It opens, potassium ions flood out, and the sodium-induced electrical potential shuts down.

In some types of epilepsy, the KCNQ potassium channels have trouble opening and shutting down runaway electrical potentials in the nerve synapse. Retigabine helps them open.

There are five different kinds of KCNQ potassium channels in the body, but only two are important in epilepsy and tinnitus: KCNQ2 and KCNQ3. The problem with retigabine is that it acts on other KCNQ potassium channels as well, and that’s why it has so many unwanted side effects.

Tzingounis and Tzounopoulos first tested SF0034 in neurons, and found that it was more selective than retigabine. It seemed to open only KCNQ2 and KCNQ3 potassium channels, not affecting KCNQ 4 or 5. It was more effective than retigabine at preventing seizures in animals, and it was also less toxic.The results are promising, both for research and for medicine. SciFluor now plans to start FDA trials with SF0034, to see whether it is safe and effective in people. Treating epilepsy is the primary goal, but tinnitus can be similarly debilitating, and sufferers would welcome a decent treatment.

Tzingounis is pleased as well. “This [SF0034] gives me another tool, and a better tool, to dissect the function of these channels,” he says. “We need to find solutions for kids – and adults – with this problem.”


“Yo, you good ma?”

You can see the larger tumor better when she’s standing… look at my good girl still guarding me like she should.

Was talking to my friend Kelsey about one time I had a seizure in a movie theater lobby. Lucky opened my purse and got my shot, she got the attention of people around so I was caught by a kind gentleman whom she showed the shot to and showed him to put it in my arm. When I woke up, I was in a haze, as normal. Sometimes you forget everything. She gave me my wallet so I knew who I was. She lay on me and waited for me to be okay. Finally it came back to me and I said “Lucky!” She jumped up and smiled/panted, wagging her little nubby butt, and licked my face. THAT’S a service dog. 

THIS service dog has cancer. This amazing creature is very sick and needs surgery and medications I can’t afford. Please read the full story here.

Thank you to everyone blogging and sharing, caring and donating. You’re amazing!

Anneliese Michel  was known by everyone as a religious and kind young girl. She was 16 when she was diagnosed with temporal lobe epilepsy after a severe convulsion and was medicated but things started changing after that. Michel started having hallucinations and hearing voices that would tell her she was “ damned. “ Psychiatric treatment did not help and Michel worsening condition started being attributed to demonic possession when she started having violent reactions to the sight of a crucifix and other sacred objects. Instead of pursuing more medical help her family consulted several priests asking for an exorcism.

On September 24, 1975, priest Arnold Renz preformed his first exorcism on Michel in secrecy with total approval of  Bishop Josef Stangl. Michel’s family completely abandoned other treatment options and focused only on the exorcisms that would last for hours, several days a week. Michel’s condition worsened and she stopped eating. On July 1, 1976, after almost 10 months of exorcism sessions, Anneliese Michel died in her own home.

The autopsy showed signs of starvation and dehydration, she was extremely malnourished weighing only 30 kg.

Michel’s parents and priests Ernst Alt and Arnold Renz were charged with negligentg homicide, the priests were sentenced to pay a fine whole Michel’s parents were left free of any accusations.
Radical Neurodivergence Speaking: Things I cannot do--because I'm epileptic

Roughly 30% of autistic people are also epileptic, so it’s impossible to make a product or event autism friendly without also making it safe for epilepsy.

Want to make the world a safer place for autistic people? Disable the flash on your camera. Turn down (or better yet, off) your music. And above all stop using strobe lights in any context.

Study adds new evidence linking brain mutation to autism, epilepsy and other neuro disorders

Findings, published in Nature Communications, reveal the extent a mutation associated with autism and epilepsy plays in impairing a biochemical process in the brain. The study, led by University of Bristol researchers, could provide a new target for treating neurological disorders.

The brain contains billions of nerve cells which communicate via the release of chemicals at connections called synapses. Each nerve cell can have thousands of synaptic connections to hundreds of other nerve cells. The protein Synapsin 1a plays a key role in regulating how synapses operate by regulating the amount of chemical transmission.

SUMOylation is a chemical process in which a protein called SUMO is attached to a target protein and modifies its function. Researchers studying SUMOylation in the brain have shown that synapsin 1a is a target protein for SUMOylation. They also found that a mutation called A548T in synapsin 1a, which has already been associated with autism and epilepsy, reduces synapsin 1a SUMOylation and interferes with its ability to function, causing impaired synaptic function that may contribute to neurological disease.

Jeremy Henley, Professor of Molecular Neuroscience in the University’s School of Biochemistry in the Faculty of Medical and Veterinary Sciences and the study’s lead author, said: “These results show the extent and how critical the role synaptic proteins regulated by SUMOylation play in neurological disorders. Importantly, they provide further evidence for SUMO modification of synaptic proteins in health and disease.”

How Music Can Help People with Epilepsy

The brains of people with epilepsy appear to react to music differently from the brains of those who do not have the disorder, a finding that could lead to new therapies to prevent seizures.

The research “Music and the Brain: Can Music Help People with Epilepsy?” will be presented at the American Psychological Association’s 123rd Annual Convention in the Metro Toronto Convention Centre, Toronto, Canada on Moday August 9 2015 at 9 am.

Image: The researchers found significantly higher levels of brainwave activity in participants when they were listening to music. More important, brainwave activity in people with epilepsy tended to synchronize more with the music, especially in the temporal lobe, than in people without epilepsy. Image is for illustrative purposes only.

vigilpanty asked:

Hi, I'm super glad to hear Kiba's doing so good with it all! My dog, Tucker, has epilepsy as well, so I can kinda relate. I wish you guys all the best! ❤🐶

It’s so scary isn’t it?? I’m afraid we’re going to become complacent and “forget” how scary it is , and then he’ll have one and scare us all over again. I hope your pup is doing okay on his meds. We’re going to enjoy every seizure-free day that we can!

Epilepsy has been found to reduce the generation of new neurons

Amanda Sierra and Juan Manuel Encinas, Ikerbasque researchers at the Achucarro centre (Achucarro Basque Center for Neuroscience) have discovered a new property of hippocampal neural stem cells by using an epilepsy model in genetically modified mice. The work has been published by the prestigious “Cell Stem Cell” journal, the main international reference in stem cell research.

The mission of neural stem cells located in the hippocampus, one of the main regions of the brain, is to generate new neurons during the adult life of mammals, including human beings, of course, and their function is to participate in certain types of learning and responses to anxiety and stress. Using an epilepsy model in genetically modified mice, the researchers have discovered that hippocampal neural stem cells stop generating new neurons and are turned into reactive astrocytes, a cell type that promotes inflammation and alters communication between neurons.

This research work has also made it possible to confirm the hypothesis in a previous piece of research by these researchers; this hypothesis established that even though neuronal hyperexcitation does not go as far as to cause convulsions, it does induce the massive activation of neural stem cells and their resulting premature exhaustion; as a result, neurogenesis (generation of new neurons) in the hippocampus ends up chronically reduced.

Juan Manuel Encinas, who is leading the study, highlights the fact that “this discovery has enabled us to gain a better understanding about how neural stem cells function. We have shown that in addition to generating neurons and astrocytes, neural stem cells in the adult hippocampus can generate reactive astrocytes following an epileptic seizure”.

Even though the work has been carried out on experimental animals, this discovery has clear implications in clinical practice and in the quest for new therapies for epilepsy given that the generation of new neurons (neurogenesis) is a process that is negatively affected in epileptic seizures located in the hippocampus. “If we can manage to preserve the population of neural stem cells and their capacity to generate new neurons in humans, it may be possible to prevent the development of certain symptoms associated with epilepsy and very likely to mitigate the damage that is caused in the hippocampus,” pointed out Juan Manuel Encinas.

(Image: Shutterstock)