How to Keep Calm!

1. Remind yourself that worrying doesn’t stop things happening. Things will happen – or not happen –anyway. Don’t forget to breath! :)

2. Recognise that “What ifs” don’t usually help with problem solving. It’s better to use logic, and brain storm for solutions. Take control of your emotions by using rational thinking. Take a step back, solve the problem.

3. Motivate yourself by something other than worrying. Take a break and do something fun, and then go back to your work again. That positive approach will reap more benefits. Play some rugby, or go to the gym, American football, whatever it is, just keep yourself busy but take a break from worrying.

4. Face your fears – and do the things that you worry about. The thought is often much worse than the actual thing you fear. It’s all in your head!

5. Ask yourself “What’s the worst thing that could happen?” Then, “What are the chances that it will happen? Then “Will you survive it, if it happens, in the end?”. Usually, that helps to move us from an extreme and irrational way of thinking to a more realistic, and reasonable way if thinking.

6. Teach yourself a range of relaxation strategies – and then concentrate on them instead of on your different fears. Or, adopt a mindful approach – and keep your focus on “right now”.

i feel a lot of psychiatrists don’t mention this crucial piece of information, so here it is: if you’re on a psychiatric medication (or other prescriptions meds), chances are you should NOT be eating grapefruit.  you should also be careful to not eat other citrus fruits in large quantities.

Grapefruit juice blocks special enzymes in the wall of the small intestine that actually destroys many medications and prevents their absorption into the body. Thus, smaller amounts of the drugs get into the body than are ingested. When the action of this enzyme is blocked, more of the drugs get into the body and the blood levels of these medications increase. This can lead to toxic side effects from the medications.

-medicinenet.com

the medications vary, but include especially psychiatric medications, and those mentioned here:

The list of medications that can interact with grapefruit is long and includes commonly prescribed medicines that fight infections, reduce cholesterol, treat high blood pressure and treat heart problems.

-mayo clinic

some more information from the cbc:

David Bailey, a clinical pharmacologist at the Lawson Health Research Institute in London, Ont., discovered the interaction between grapefruit and certain medications more than 20 years ago. Since then, he said, the number of drugs with the potential to interact has jumped to more than 85.

Of the 85 known drugs that interact with grapefruit, 43 can have serious side-effects, including sudden death, acute kidney failure, respiratory failure, gastrointestinal bleeding and bone marrow suppression in people with weakened immune systems.

The authors noted that all sources of grapefruit — the whole fruit or 200 mL of grapefruit juice — and other citrus fruit such as Seville oranges (often used in marmalade), limes and pomelos can lead to drug interactions.

-cbc

& lastly, nps has a non-complete list of medications that do interact with grapefruit, that link is here.

if you’re on medication, especially psychiatric medication, i highly recommend you google your specific drug to find out about grapefruit interactions, or phone your pharmacy and ask the pharmacists.

please feel free to reblog, unfortunately a lot of doctors don’t warn patients about the danger of grapefruit and i want people to be aware.

anonymous asked:

"We left school at an early age"(!) I'm a (sort of) newbie. What was going on at the time?

Are you talking about this?

Liam was just confused about whether psychiatry is a word, and Harry joked that they left school at an early age (which they did, in order to be One Direction – like, none of them went to uni, Harry stopped going to school at 16, etc.) as the explanation for why Liam was confused. Just a joke! Not part of anything bigger that was happening.

“Muted Affection”

Threadless has teamed up with Peanuts and asked the artist community to put our own spin on the creations of Charles M. Schulz.

I come from a musical family and I know a muted trombone when I hear it, so I just assumed Charlie Brown’s parents were jazz musicians who were too busy practicing to pay attention to him.

Make it a t-shirt on Threadless   Facebook   Tumblr

How well your immune system can fight infection may depend on your personality, new research, led by Professor Kavita Vedhara from the University of Nottingham , has found.

Extroverts, individuals who we would expect to be exposed to more infections as a result of their socially orientated nature, are likely to have stronger immune systems to deal effectively with infection than those who are more cautious, the findings showed.

“Our results indicated that ‘extraversion’ was significantly associated with an increased expression of pro-inflammatory genes and that ‘conscientiousness’ was linked to a reduced expression of pro-inflammatory genes,” said Professor Kavita Vedhara from the University of Nottingham.

The findings support long-observed associations between aspects of human character, physical health and longevity.

The researchers examined the relationship between certain personality traits and the expression of genes which can affect our health by controlling the activity of our immune systems in a group of 121 ethnically diverse and healthy adults.

The study used highly-sensitive microarray technology to examine the relationship between five major human personality traits and two groups of genes active in human white blood cells (leukocytes) — one involving inflammation and another involving antiviral responses and antibodies.

The participants completed a personality test which measures extraversion, neuroticism, openness, agreeableness and conscientiousness.

“Individuals who we would expect to be exposed to more infections as a result of their socially orientated nature (ie, extroverts) appear to have immune systems that we would expect can deal effectively with infection,” Vedhara pointed out.

“While individuals who may be less exposed to infections because of their cautious/conscientious dispositions have immune systems that may respond less well,” she added.

Credit: x

Post-Traumatic Stress Disorder Linked to Accelerated Aging

In recent years, public health concerns about post-traumatic stress disorder (PTSD) have risen significantly, driven in part by affected military veterans returning from conflicts in the Middle East and elsewhere. PTSD is associated with number of psychological maladies, among them chronic depression, anger, insomnia, eating disorders and substance abuse.

Writing in the May 7 online issue of American Journal of Geriatric Psychiatry, researchers at University of California, San Diego School of Medicine and Veterans Affairs San Diego Healthcare System suggest that people with PTSD may also be at risk for accelerated aging or premature senescence.

“This is the first study of its type to link PTSD, a psychological disorder with no established genetic basis, which is caused by external, traumatic stress, with long-term, systemic effects on a basic biological process such as aging,” said Dilip V. Jeste, MD, Distinguished Professor of Psychiatry and Neurosciences and director of the Center on Healthy Aging and Senior Care at UC San Diego, who is the senior author of this study.

Researchers had previously noted a potential association between psychiatric conditions, such as schizophrenia and bipolar disorder, and acceleration of the aging process. Jeste and colleagues determined to see if PTSD might show a similar association by conducting a comprehensive review of published empirical studies relevant to early aging in PTSD, covering multiple databases going back to 2000.

There is no standardized definition of what constitutes premature or accelerated senescence. For guidance, the researchers looked at early aging phenomena associated with non-psychiatric conditions, such as Hutchinson-Gilford progeria syndrome, HIV infection and Down’s syndrome. The majority of evidence fell into three categories: biological indicators or biomarkers, such as leukocyte telomere length (LTL), earlier occurrence or higher prevalence of medical conditions associated with advanced age and premature mortality.

In their literature review, the UC San Diego team identified 64 relevant studies; 22 were suitable for calculating overall effect sizes for biomarkers, 10 for mortality.  

All six studies looking specifically at LTL found reduced telomere length in persons with PTSD. Leukocytes are white blood cells. Telomeres are stretches of protective, repetitive nucleotide sequences at the ends of chromosomes. These sequences shorten with every cell replication and are considered a strong measure of the aging process in cells.  

The scientists also found consistent evidence of increased pro-inflammatory markers, such as C-reactive protein and tumor necrosis factor alpha, associated with PTSD.

A majority of reviewed studies found increased medical comorbidity of PTSD with several targeted conditions associated with normal aging, including cardiovascular disease, type 2 diabetes, gastrointestinal ulcer disease and dementia.

Seven of 10 studies indicated a mild-to-moderate association of PTSD with earlier mortality, consistent with an early onset or acceleration of aging in PTSD.

“These findings do not speak to whether accelerated aging is specific to PTSD, but they do argue the need to re-conceptualize PTSD as something more than a mental illness,” said first author James B. Lohr, MD, professor of psychiatry. “Early senescence, increased medical morbidity and premature mortality in PTSD have implications in health care beyond simply treating PTSD symptoms. Our findings warrant a deeper look at this phenomenon and a more integrated medical-psychiatric approach to their care.”

Barton Palmer, PhD, professor of psychiatry and a coauthor of the study, cautioned that “prospective longitudinal studies are needed to directly demonstrate accelerated aging in PTSD and to establish underlying mechanisms.”

Pictured: Human chromosomes shown in blue, with telomeres appearing as yellow pinpoints. Image courtesy of the National Institutes of Health.